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Listar por autor "Juárez, Carlos"
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Anticonvulsant-induced Toxic Epidermal Necrolysis: Monitoring the Immunologic Response.
Leyva-Fernández, Laura; Torres-Jaén, María Josefa; Posadas, Sinforiano Jose; Blanca, Miguel; Besso, Guillermo; O'Valle, Francisco; García del Moral, Raimundo; Santamaría, Luis F; Juárez, Carlos[et al.] (Elsevier BV, 2000)Background Toxic epidermal necrolysis is a severe reaction with skin involvement induced by different drugs and other agents. The mechanisms implicated in the induction of the reaction are poorly understood. Objective To ... -
Expression of the skin-homing receptor in peripheral blood lymphocytes from subjects with nonimmediate cutaneous allergic drug reactions
Blanca, Miguel; Posadas, Sinforiano Jose; Torres-Jaén, María Josefa; Leyva-Fernández, Laura; Mayorga Mayorga, Cristobalina; González, Lourdes; Juárez, Carlos; Fernández, Javier; Santamaría, Luis F[et al.] (Wiley, 2000)Background: In nonimmediate cutaneous reactions to drugs, the skin is the organ most frequently involved, and T lymphocytes may play a relevant role. T cells related to skin immune responses express the cutaneous ... -
In vitro T-cell responses to beta-lactam drugs in immediate and nonimmediate allergic reactions.
Luque, Inmaculada; Leyva-Fernández, Laura; Torres-Jaén, María Josefa; Rosal, Manuel; Mayorga Mayorga, Cristobalina; Segura, José Manuel; Blanca, Miguel; Juárez, Carlos[et al.] (Wiley, 2008)Background: Beta-Lactam drugs may induce both cellular and humoral allergic reactions, and there is evidence that T cells play an important role in the pathogenesis of these reactions. The aim of this work was to assess ... -
Subjects with allergic reactions to drugs show in vivo polarized patterns of cytokine expression depending on the chronology of the clinical reaction
Posadas, Sinforiano Jose; Leyva-Fernández, Laura; Torres-Jaén, María Josefa; Rodríguez-Bada, José Luis; Bravo, Inmaculada; Rosal, Manuel; Fernández, Javier; Juárez, Carlos; Blanca, Miguel[et al.] (Elsevier, 2000-10)Background: The mechanisms involved in adverse drug reactions with an immunologic basis (ADRIB) can be antibody dependent, mainly IgE or T cell dependent (sensitized T cells). These mechanisms are regulated by a number of ...